Activation of β2 adrenergic receptor signaling modulates inflammation: a target limiting the progression of kidney diseases
Author:
Funder
Ewha Womans University
Publisher
Springer Science and Business Media LLC
Subject
Organic Chemistry,Drug Discovery,Molecular Medicine
Link
http://link.springer.com/content/pdf/10.1007/s12272-020-01280-9.pdf
Reference107 articles.
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3. Anderson R, Visser SS, Ramafi G, Theron AJ (2000) Accelerated resequestration of cytosolic calcium and suppression of the pro-inflammatory activities of human neutrophils by CGS 21680 in vitro. Br J Pharmacol 130:717–724. https://doi.org/10.1038/sj.bjp.0703344
4. Anderson R, Theron AJ, Steel HC, Durandt C, Tintinger GR, Feldman C (2014) The beta-2-adrenoreceptor agonists, formoterol and indacaterol, but not salbutamol, effectively suppress the reactivity of human neutrophils in vitro. Mediators Inflamm 2014:1–9. https://doi.org/10.1155/2014/105420
5. Arif E, Solanki AK, Srivastava P, Rahman B, Fitzgibbon WR, Deng P, Budisavljevic MN, Baicu CF, Zile MR, Megyesi J, Janech MG, Kwon S-H, Collier J, Schnellmann RG, Nihalani D (2019) Mitochondrial biogenesis induced by the β2-adrenergic receptor agonist formoterol accelerates podocyte recovery from glomerular injury. Kidney Int 96:656–673. https://doi.org/10.1016/j.kint.2019.03.023
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