Orphan Receptor TR3 Enhances p53 Transactivation and Represses DNA Double-Strand Break Repair in Hepatoma Cells under Ionizing Radiation-Reference-Cited by-同舟云学术

Orphan Receptor TR3 Enhances p53 Transactivation and Represses DNA Double-Strand Break Repair in Hepatoma Cells under Ionizing Radiation

Published:2011-08-01 Issue:8 Volume:25 Page:1337-1350
ISSN:0888-8809
Container-title:Molecular Endocrinology
language:en
Short-container-title:

Author:

Zhao Bi-xing¹,Chen Hang-zi¹,Du Xiao-dan¹,Luo Jie¹,He Jian-ping¹,Wang Rong-hao¹,Wang Yuan¹,Wu Rong¹,Hou Ru-rong²,Hong Ming³,Wu Qiao¹

Affiliation:

1. State Key Laboratory of Stress Cell Biology (B.Z., H.C., X.D., J.L., J.H., R.W., Y.W., R.W., Q.W.), School of Life Sciences, Xiamen University, Xiamen 361005, Fujian Province, China;

2. Zhongshan Hospital (R.H.), Xiamen University, Xiamen 361004, Fujian Province, China;

3. The First Affiliated Hospital of Xiamen University (M.H.), Xiamen, Fujian 361003, China

Publisher

The Endocrine Society

Subject

Endocrinology,Molecular Biology,General Medicine

Link

http://academic.oup.com/mend/article-pdf/25/8/1337/8932546/mend1337.pdf

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3. The DNA-dependent protein kinase.;Smith;Genes Dev,1999

4. The DNA-dependent protein kinase: requirement for DNA ends and association with Ku antigen.;Gottlieb;Cell,1993

5. Autophosphorylation of DNA-PKCS regulates its dynamics at DNA double-strand breaks.;Uematsu;J Cell Biol,2007

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