Rescue of the Skeletal Phenotype of Vitamin D Receptor-Ablated Mice in the Setting of Normal Mineral Ion Homeostasis: Formal Histomorphometric and Biomechanical Analyses1-Reference-Cited by-同舟云学术

Rescue of the Skeletal Phenotype of Vitamin D Receptor-Ablated Mice in the Setting of Normal Mineral Ion Homeostasis: Formal Histomorphometric and Biomechanical Analyses1

Published:1999-11-01 Issue:11 Volume:140 Page:4982-4987
ISSN:0013-7227
Container-title:Endocrinology
language:en
Short-container-title:

Author:

Amling Michael¹,Priemel Matthias²,Holzmann Timothy²,Chapin Kelli³,Rueger Johannes M.²,Baron Roland¹,Demay Marie B.³

Affiliation:

1. The Department of Cell Biology, Yale University School of Medicine (M.A., R.B.), New Haven, Connecticut 06510

2. Department of Trauma Surgery, Hamburg University School of Medicine (M.A., M.P., T.H., J.M.R.), Hamburg, Germany

3. The Endocrine Unit, Massachusetts General Hospital, Harvard Medical School (K.C., M.B.D.), Boston, Massachusetts 02114

Publisher

The Endocrine Society

Subject

Endocrinology

Link

http://academic.oup.com/endo/article-pdf/140/11/4982/10376677/endo4982.pdf

Reference23 articles.

1. Ontogeny of the 1,25-dihydroxyvitamin D3 receptor in fetal rat bone.;Johnson;J Bone Miner Res,1996

2. Vitamin D is not directly necessary for bone growth and mineralization;Underwood;Am J Physiol,1984

3. Bone histomorphometry in vitamin D-deficient rats infused with calcium and phosphorus;Weinstein;Am J Physiol,1984

4. Long-term nocturnal calcium infusions can cure rickets and promote normal mineralization in hereditary resistance to 1,25-dihydroxyvitamin D.;Balsan;J Clin Invest,1986

5. Noncollagenous bone proteins in experimental rickets in the rat.;Weintroub;Mol Cell Biochem,1987

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