Oncogenic role of miR ‐155 in anaplastic large cell lymphoma lacking the t(2;5) translocation-Reference-Cited by-同舟云学术

Oncogenic role of miR ‐155 in anaplastic large cell lymphoma lacking the t(2;5) translocation

Published:2015-04-27 Issue:4 Volume:236 Page:445-456
ISSN:0022-3417
Container-title:The Journal of Pathology
language:en
Short-container-title:J. Pathol.

Author:

Merkel Olaf¹²³,Hamacher Frank⁴,Griessl Robert⁴,Grabner Lisa²,Schiefer Ana‐Iris²,Prutsch Nicole²,Baer Constance⁵,Egger Gerda²³,Schlederer Michaela²⁶,Krenn Peter William⁴,Hartmann Tanja Nicole⁴,Simonitsch‐Klupp Ingrid²,Plass Christoph⁵,Staber Philipp Bernhard⁷,Moriggl Richard⁶⁸,Turner Suzanne D⁹³,Greil Richard⁴,Kenner Lukas²⁶¹⁰³

Affiliation:

1. Department of Translational Oncology, National Centre for Tumour Diseases (NCT) German Cancer Research Centre (DKFZ) Heidelberg Germany

2. Department of Clinical Pathology Medical University Vienna Austria

3. European Research Initiative on ALK Related Malignancies (www.erialcl.net)

4. Laboratory for Immunological and Molecular Cancer Research, Third Medical Department, Oncologic Centre Paracelsus Medical University Salzburg Austria

5. Department of Epigenomics and Cancer Risk Factors, German Cancer Research Centre (DKFZ) Heidelberg Germany

6. Ludwig Boltzmann Institute for Cancer Research Vienna Austria

7. Division of Hematology and Hemostaseology, Comprehensive Cancer Centre Vienna Medical University of Vienna 1090 Vienna Austria

8. Institute of Animal Breeding and Genetics University of Veterinary Medicine Vienna and Medical University of Vienna Austria

9. Division of Molecular Histopathology, Department of Pathology University of Cambridge UK

10. Unit of Pathology of Laboratory Animals University of Veterinary Medicine Vienna Austria

Funder

SCRI-LIMCR GmbH, Jubiläumsfond der Österreichischen Nationalbank

Fonds der Stadt Wien für innovative interdisziplinäre Krebsforschung

Austrian Science Funds (FWF)

Publisher

Wiley

Subject

Pathology and Forensic Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/path.4539

Reference48 articles.

1. The anaplastic lymphoma kinase in the pathogenesis of cancer

2. Anaplastic large-cell lymphoma

3. Activation of Mammalian Target of Rapamycin Signaling Pathway Contributes to Tumor Cell Survival in Anaplastic Lymphoma Kinase–Positive Anaplastic Large Cell Lymphoma

4. Loss of SHP1 enhances JAK3/STAT3 signaling and decreases proteosome degradation of JAK3 and NPM-ALK in ALK+ anaplastic large-cell lymphoma

5. Stat3 is required for ALK-mediated lymphomagenesis and provides a possible therapeutic target

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