KITD816VandJAK2V617Fmutations are seen recurrently in hypereosinophilia of unknown significance

Author:

Schwaab Juliana1,Umbach Roland1,Metzgeroth Georgia1,Naumann Nicole1,Jawhar Mohamad1,Sotlar Karl2,Horny Hans-Peter2,Gaiser Timo3,Hofmann Wolf-Karsten1,Schnittger Susanne4,Cross Nicholas C.P.56,Fabarius Alice1,Reiter Andreas1

Affiliation:

1. Department of Hematology and Oncology; University Hospital Mannheim; Mannheim Germany

2. Department of Pathology; Ludwig Maximilians University; Munich Germany

3. Department of Pathology; University Hospital Mannheim; Mannheim Germany

4. MLL, Munich Leukemia Laboratory; Munich Germany

5. Wessex Regional Genetics Laboratory; University of Southampton, Salisbury District Hospital; Salisbury United Kingdom

6. Faculty of Medicine; University of Southampton; Southampton United Kingdom

Funder

Deutsche José Carreras Leukämie Stiftung e.V.′ (H11/03), Germany

Publisher

Wiley

Subject

Hematology

Reference28 articles.

1. ICON: Eosinophil disorders;Valent;World Allergy Organ J,2012

2. Pathogenesis and classification of eosinophil disorders: A review of recent developments in the field;Valent;Expert Rev Hematol,2012

3. FIP1L1-PDGFR alpha, a therapeutic target for the treatment of chronic eosinophilic leukemia;Cools;Verh K Acad Geneeskd Belg,2005

4. The t(8;17)(p11;q23) in the 8p11 myeloproliferative syndrome fuses MYO18A to FGFR1;Walz;Leukemia,2005

5. Transient response to imatinib in a chronic eosinophilic leukemia associated with ins(9;4)(q33;q12q25) and a CDK5RAP2-PDGFRA fusion gene;Walz;Genes Chromosomes Cancer,2006

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