SLC26A9 stimulates CFTR expression and function in human bronchial cell lines
Author:
Publisher
Wiley
Subject
Cell Biology,Clinical Biochemistry,Physiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1002/jcp.22328/fullpdf
Reference30 articles.
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2. Modulation of calcium-dependent chloride secretion by basolateral SK4-like channels in a human bronchial cell line;Bernard;J Membr Biol,2003
3. SLC26A9 is a constitutively active, CFTR-regulated anion conductance in human bronchial epithelia;Bertrand;J Gen Physiol,2009
4. Cl− transport by cystic fibrosis transmembrane conductance regulator (CFTR) contributes to the inhibition of epithelial Na+ channels (ENaCs) in Xenopus oocytes co-expressing CFTR and ENaC;Briel;J Physiol,1998
5. Slc26a9 is inhibited by the R-region of CFTR via the STAS domain;Chang;J Biol Chem,2009a
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