The solute carrier family 26 member 9 modifies rapidly progressing cystic fibrosis associated with homozygous F508del CFTR mutation-Reference-Cited by-同舟云学术

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The solute carrier family 26 member 9 modifies rapidly progressing cystic fibrosis associated with homozygous F508del CFTR mutation

Published:2024-07 Issue: Volume:561 Page:119765
ISSN:0009-8981
Container-title:Clinica Chimica Acta
language:en
Short-container-title:Clinica Chimica Acta

Author:

Luo Shiyu^ORCID,Rollins Stuart,Schmitz-Abe Klaus,Tam Amy,Li Qifei,Shi Jiahai,Lin Jasmine,Wang Ruobing,Agrawal Pankaj B.

Funder

National Cancer Institute

National Institute on Drug Abuse

National Heart, Lung, and Blood Institute

National Institute of Neurological Disorders and Stroke

National Institute of Mental Health

National Human Genome Research Institute

National Institutes of Health

Publisher

Elsevier BV

Reference54 articles.

1. Cystic fibrosis: a review;Ong;J. Am. Med. Assoc.,2023

2. Gene therapy for cystic fibrosis: recent advances and future prospects;Lomunova;Acta Nat.,2023

3. Year-to-year changes in lung function in individuals with cystic fibrosis;Liou;J. Cyst. Fibros.,2010

4. The changing epidemiology of cystic fibrosis: incidence, survival and impact of the CFTR gene discovery;Scotet;Genes (Basel),2020

5. Heritability of lung disease severity in cystic fibrosis;Vanscoy;Am. J. Respir. Crit. Care Med.,2007

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