The Cag pathogenicity island and interaction between TLR 2/ NOD 2 and NLRP 3 regulate IL ‐1β production in Helicobacter pylori infected dendritic cells-Reference-Cited by-同舟云学术

The Cag pathogenicity island and interaction between TLR 2/ NOD 2 and NLRP 3 regulate IL ‐1β production in Helicobacter pylori infected dendritic cells

Published:2013-07-25 Issue:10 Volume:43 Page:2650-2658
ISSN:0014-2980
Container-title:European Journal of Immunology
language:en
Short-container-title:Eur. J. Immunol.

Author:

Kim Dong‐Jae¹²,Park Jong‐Hwan¹²³,Franchi Luigi¹,Backert Steffen⁴,Núñez Gabriel¹

Affiliation:

1. Department of Pathology and Comprehensive Cancer Center University of Michigan Medical School Ann Arbor MI USA

2. World Class Institute Korea Research Institute of Bioscience and Biotechnology, Ochang‐Eup Cheongwon‐Gun Choongbuk Korea

3. Department of Biochemistry College of Medicine, Konyang University Daejeon Korea

4. Institute of Medical Microbiology Otto von Guericke University Magdeburg, Leipziger Magdeburg Germany

Funder

National Institutes of Health

National Research Foundation of Korea

Korean Government

World Class Institute (WCI) program

National Research Foundation of Korea (NRF)

Ministry of Education, Science and Technology of Korea (MEST)

Research Career Development Award from the Crohn's and Colitis Foundation of America

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/eji.201243281

Reference35 articles.

1. The role of pattern-recognition receptors in innate immunity: update on Toll-like receptors

2. Intracellular NOD-like Receptors in Host Defense and Disease

3. An essential role for NOD1 in host recognition of bacterial peptidoglycan containing diaminopimelic acid

4. Nod1 Detects a Unique Muropeptide from Gram-Negative Bacterial Peptidoglycan

5. Nod2 Is a General Sensor of Peptidoglycan through Muramyl Dipeptide (MDP) Detection

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