Eruca sativaand its Flavonoid Components, Quercetin and Isorhamnetin, Improve Skin Barrier Function by Activation of Peroxisome Proliferator-Activated Receptor (PPAR)-α and Suppression of Inflammatory Cytokines

Author:

Kim Bora1,Choi Yoon-E2,Kim Hyun-Soo3

Affiliation:

1. Skin & Bio Research; Ellead Co., Ltd.; 325, Hwangsaeul-ro, Bundang-gu Seongnam-si Gyeonggi-do 463-824 Republic of Korea

2. LED Agri-bio Fusion Technology Research Center; Chonbuk National University; 79, Gobong-ro Iksan-si Jeollabuk-do 570-752 Republic of Korea

3. Department of Food Science and Industry; Jungwon University; 85, Munmu-ro, Geosan-eup Geosan-gun Chungcheongbuk-do 367-805 Republic of Korea

Publisher

Wiley

Subject

Pharmacology

Reference33 articles.

1. Human epidermal keratinocytes undergo (−)-epigallocatechin-3-gallate-dependent differentiation but not apoptosis;Balasubramanian;Carcinogenesis,2005

2. Myelin alters the inflammatory phenotype of macrophages by activating PPARs;Bogie;Acta Neuropathol Commun,2013

3. The integration of physiologically-targeted skin care in the management of atopic dermatitis: focus on the use of a cleanser and moisturizer system incorporating a ceramide precursor, filaggrin degradation products, and specific “skin-barrier-friendly” excipients;Del Rosso;J Drugs Dermatol,2013

4. Interleukin-10-mediated inhibition of free radical generation in macrophages;Dokka;Am J Physiol Lung Cell Mol Physiol,2001

5. Lipid and protein structures in the permeability barrier of mammalian epidermis;Downing;J Lipid Res,1992

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