TAK1 inhibition improves myoblast differentiation and alleviates fibrosis in a mouse model of Duchenne muscular dystrophy-Reference-Cited by-同舟云学术

TAK1 inhibition improves myoblast differentiation and alleviates fibrosis in a mouse model of Duchenne muscular dystrophy

Published:2020-11-25 Issue:1 Volume:12 Page:192-208
ISSN:2190-5991
Container-title:Journal of Cachexia, Sarcopenia and Muscle
language:en
Short-container-title:Journal of Cachexia, Sarcopenia and Muscle

Author:

Xu Dengqiu¹,Li Sijia¹,Wang Lu¹,Jiang Jingwei¹,Zhao Lei²,Huang Xiaofei¹,Sun Zeren¹,Li Chunjie¹,Sun Lixin¹,Li Xihua²,Jiang Zhenzhou¹³^ORCID,Zhang Luyong¹⁴³

Affiliation:

1. Jiangsu Key Laboratory of Drug Screening China Pharmaceutical University Nanjing China

2. Department of Neurology Children's Hospital of Fudan University Shanghai China

3. Key Laboratory of Drug Quality Control and Pharmacovigilance China Pharmaceutical University Nanjing China

4. Center for Drug Research and Development Guangdong Pharmaceutical University Guangzhou China

Funder

Joint Fund of the National Natural Science Foundation of China and the Karst Science Research Center of Guizhou Province

Publisher

Wiley

Subject

Physiology (medical),Orthopedics and Sports Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcsm.12650

Reference51 articles.

1. Evidence-based path to newborn screening for duchenne muscular dystrophy

2. Animal models of Duchenne muscular dystrophy: from basic mechanisms to gene therapy

3. Progress toward Gene Therapy for Duchenne Muscular Dystrophy

4. Detection of long‐term progression of myocardial fibrosis in Duchenne muscular dystrophy in an affected family: a cardiovascular magnetic resonance study;Walcher T;Eur J Radiol,2011

5. Fibrosis Rescue Improves Cardiac Function in Dystrophin-Deficient Mice and Duchenne Patient–Specific Cardiomyocytes by Immunoproteasome Modulation

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