A Tale of Two Complications of Obesity: NASH and Hepatocellular Carcinoma
Author:
Affiliation:
1. Division of Gastroenterology and Hepatology Stanford University Stanford CA
2. Division of Medical Oncology, Departments of Medicine and Pathology Stanford University Stanford CA
Funder
National Institutes of Health
National Cancer Institute
Publisher
Wiley
Subject
Hepatology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/hep.30649
Reference8 articles.
1. Hepatocellular carcinomas in patients with metabolic syndrome often develop without significant liver fibrosis: A pathological analysis
2. Systematic review with meta-analysis: risk of hepatocellular carcinoma in non-alcoholic steatohepatitis without cirrhosis compared to other liver diseases
3. Obesity Drives STAT-1-Dependent NASH and STAT-3-Dependent HCC
4. Hepatic Oxidative Stress Promotes Insulin-STAT-5 Signaling and Obesity by Inactivating Protein Tyrosine Phosphatase N2
5. Opposing roles of STAT1 and STAT3 in T cell–mediated hepatitis: regulation by SOCS
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