Obesity Drives STAT-1-Dependent NASH and STAT-3-Dependent HCC-Reference-Cited by-同舟云学术

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Obesity Drives STAT-1-Dependent NASH and STAT-3-Dependent HCC

Published:2018-11 Issue:5 Volume:175 Page:1289-1306.e20
ISSN:0092-8674
Container-title:Cell
language:en
Short-container-title:Cell

Author:

Grohmann Marcus,Wiede Florian,Dodd Garron T.,Gurzov Esteban N.,Ooi Geraldine J.,Butt Tariq,Rasmiena Aliki A.,Kaur Supreet,Gulati Twishi,Goh Pei K.,Treloar Aislinn E.,Archer Stuart,Brown Wendy A.,Muller Mathias,Watt Matthew J.,Ohara Osamu,McLean Catriona A.,Tiganis Tony

Funder

NHMRC of Australia

Cancer Council Victoria

Association for International Cancer Research

Austrian Science Fund

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference63 articles.

1. Non-alcoholic fatty liver disease contributes to hepatocarcinogenesis in non-cirrhotic liver: a clinical and pathological study;Alexander;J. Gastroenterol. Hepatol.,2013

2. Essential role of STAT3 in the control of the acute-phase response as revealed by inducible gene inactivation [correction of activation] in the liver;Alonzi;Mol. Cell. Biol.,2001

3. Mitochondrial H2O2 emission and cellular redox state link excess fat intake to insulin resistance in both rodents and humans;Anderson;J. Clin. Invest.,2009

4. Liver lipocalin 2 expression in severely obese women with non alcoholic fatty liver disease;Auguet;Exp. Clin. Endocrinol. Diabetes,2013

5. Hepatocellular carcinoma in non-alcoholic fatty liver disease: an emerging menace;Baffy;J. Hepatol.,2012

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