Lewis Rat Model of Experimental Autoimmune Encephalomyelitis
Author:
Affiliation:
1. Department of Neurology, St. Josef Hospital, Ruhr‐University Bochum, Bochum Germany
Publisher
Wiley
Subject
General Medicine
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/cpns.36
Reference61 articles.
1. The N-terminal domain of the myelin oligodendrocyte glycoprotein (MOG) induces acute demyelinating experimental autoimmune encephalomyelitis in the Lewis rat
2. The rapid isolation of clonable antigen-specific T lymphocyte lines capable of mediating autoimmune encephalomyelitis
3. Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells
4. Myelin Oligodendrocyte Glycoprotein–specific T Cell Receptor Transgenic Mice Develop Spontaneous Autoimmune Optic Neuritis
5. Clinical and histological findings in proteolipid protein-induced experimental autoimmune encephalomyelitis (EAE) in the lewis rat. Distribution of demyelination differs from that in EAE induced by other antigens
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