Cornuside ameliorated experimental autoimmune encephalomyelitis by limiting the recruitment of CD4+ T lymphocytes in the spinal cord
Author:
Affiliation:
1. The Second Affiliated Hospital of Zhejiang Chinese Medical University, China
2. Zhejiang Rehabilitation Medical Center, China
3. Zhejiang Chinese Medical University, China
Publisher
FapUNIFESP (SciELO)
Link
http://www.scielo.br/scielo.php?script=sci_pdf&pid=S1984-82502022000100695&tlng=en
Reference35 articles.
1. Cell surface levels of endothelial ICAM-1 influence the transcellular or paracellular T-cell diapedesis across the blood-brain barrier;Abadier M;Eur J Immunol,2015
2. T lymphocytes migrate upstream after completing the leukocyte adhesion cascade;Anderson NR;Cell Adh Migr.,2019
3. Tumor necrosis factor alpha (TNF-alpha) receptor-II is required for TNF-alpha-induced leukocyte-endothelial interaction in vivo;Chandrasekharan UM;Blood,2007
4. Cornuside suppresses lipopolysaccharide-induced inflammatory mediators by inhibiting nuclear factor-kappa B activation in RAW 264.7 macrophages;Choi YH;Biol Pharm Bull,2011
5. Experimental autoimmune encephalomyelitis (EAE) as a model for multiple sclerosis (MS);Constantinescu CS;Br J Pharmacol,2011
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