Elevated middle cerebral artery peak systolic velocity without fetal anemia in a case of homozygous α–thalassemia-1
Author:
Publisher
Wiley
Subject
Genetics (clinical),Obstetrics and Gynecology
Reference10 articles.
1. α-Thalassemia: Hb H Disease and Hb Barts Hydrops Fetalis
2. Homozygous α-thalassaemia and hypospadias - common aetiology or incidental association?
3. Inhomogeneity of Hemoglobin. VI. The Minor Hemoglobin Components of Cord Blood
4. Diagnosis, surveillance, and treatment of the anemic fetus using middle cerebral artery peak systolic velocity measurement
5. First application of denaturing high-performance liquid chromatography (DHPLC) for prenatal diagnosis of genetic disease
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1. Elevated fetal middle cerebral artery peak systolic velocity in diabetes type 1 patient: a case report;Case Reports in Perinatal Medicine;2021-01-01
2. Comparison of Resistance Index and Peak Systolic Velocity of Fetal Middle Cerebral Artery between Normal and Fetuses with Mutated Beta-Thalassemia Gene;World Family Medicine Journal/Middle East Journal of Family Medicine;2018-01
3. Prenatal ultrasound monitoring of homozygous α0-thalassemia-induced fetal anemia;Best Practice & Research Clinical Obstetrics & Gynaecology;2017-02
4. Sonographic Markers of Fetal α-Thalassemia Major;Journal of Ultrasound in Medicine;2015-02
5. Elevated middle cerebral artery peak systolic velocity as a prenatal manifestation of heterozygous type 2 alpha-thalassemia;European Journal of Obstetrics & Gynecology and Reproductive Biology;2013-04
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