Frataxin deficiency alters gene expression in Friedreich ataxia derived IPSC‐neurons and cardiomyocytes-Reference-Cited by-同舟云学术

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Frataxin deficiency alters gene expression in Friedreich ataxia derived IPSC‐neurons and cardiomyocytes

Published:2022-11-11 Issue:1 Volume:11 Page:
ISSN:2324-9269
Container-title:Molecular Genetics & Genomic Medicine
language:en
Short-container-title:Molec Gen & Gen Med

Author:

Angulo Mariana B.¹²^ORCID,Bertalovitz Alexander¹³,Argenziano Mariana A.¹,Yang Jiajia¹²,Patel Aarti³,Zesiewicz Theresa⁴,McDonald Thomas V.¹²³^ORCID

Affiliation:

1. Heart Institute, Morsani College of Medicine, University of South Florida Tampa Florida USA

2. Department of Molecular Pharmacology & Physiology Morsani College of Medicine, University of South Florida Tampa Florida USA

3. Department of Medicine (Cardiology) Morsani College of Medicine, University of South Florida Tampa Florida USA

4. Department of Neurology Morsani College of Medicine, University of South Florida Tampa Florida USA

Publisher

Wiley

Subject

Genetics (clinical),Genetics,Molecular Biology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/mgg3.2093

Reference68 articles.

1. Iron-Dependent Self-Assembly of Recombinant Yeast Frataxin: Implications for Friedreich Ataxia

2. Bacterial frataxin CyaY is the gatekeeper of iron-sulfur cluster formation catalyzed by IscS

3. The Friedreich ataxia GAA repeat expansion mutation induces comparable epigenetic changes in human and transgenic mouse brain and heart tissues

4. Generation of a Friedreich’s Ataxia patient-derived iPSC line USFi001-A

5. Emerging therapies in Friedreich's ataxia

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1. Deciphering the ferroptosis pathways in dorsal root ganglia of Friedreich ataxia models. The role of LKB1/AMPK, KEAP1, and GSK3β in the impairment of the NRF2 response;Redox Biology;2024-10

2. Intracellular calcium dysregulation in heart and brain diseases: Insights from induced pluripotent stem cell studies;Journal of Neuropathology & Experimental Neurology;2024-07-13

3. Deciphering the ferroptosis pathways in dorsal root ganglia of Friedreich ataxia models. The role of LKB1/AMPK, KEAP1, and GSK3β in the impairment of the NRF2 response;2024-05-10

4. One-to-one Benefit provided by Antioxidants to cultured skin Fibroblasts from Friedreich Ataxia patients;2024-04-28

5. A multi-faceted approach to unravel coding and non-coding gene fusions and target chimeric proteins in ataxia;Journal of Biomolecular Structure and Dynamics;2024-02-27

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