Overactivation of the nuclear factor (erythroid-derived 2)-like 2-antioxidant response element pathway in hepatocytes decreases hepatic ischemia/reperfusion injury in mice-Reference-Cited by-同舟云学术

Overactivation of the nuclear factor (erythroid-derived 2)-like 2-antioxidant response element pathway in hepatocytes decreases hepatic ischemia/reperfusion injury in mice

Published:2015-12-28 Issue:1 Volume:22 Page:91-102
ISSN:1527-6465
Container-title:Liver Transplantation
language:en
Short-container-title:Liver Transpl

Author:

Lee Lung-Yi¹,Harberg Calvin¹,Matkowskyj Kristina A.²³,Cook Shelly²,Roenneburg Drew¹,Werner Sabine⁴,Johnson Jeffrey⁵⁶⁷⁸,Foley David P.¹⁹

Affiliation:

1. Departments of Surgery; University of Wisconsin; Madison WI

2. Departments of Pathology and Laboratory Medicine, School of Medicine and Public Health; University of Wisconsin; Madison WI

3. Pathology Services, William S. Middleton Memorial Hospital, U.S. Department of Veterans Affairs; Madison WI

4. Department of Biology, Institute of Molecular Health Sciences, Swiss Federal Institute of Technology; Zurich Switzerland

5. Divisions of Pharmaceutical Sciences, University of Wisconsin; Madison WI

6. Divisions of Molecular and Environmental Toxicological Center, University of Wisconsin; Madison WI

7. Center for Neuroscience, University of Wisconsin; Madison WI

8. Waisman Center, University of Wisconsin; Madison WI

9. Surgical Services, William S. Middleton Memorial Hospital, U.S. Department of Veterans Affairs; Madison WI

Funder

Clinical and Translational Science Award (CTSA)

National Institutes of Health Center for Advancing Translational Sciences (NCATS) (D.P.F.)

American Society of Transplant Surgeons-Pfizer Mid-Level Faculty Award (D.P.F.)

National Institutes of Health (L.-Y.L.)

Swiss National Science Foundation (S.W.)

Publisher

Wiley

Subject

Transplantation,Hepatology,Surgery

Reference33 articles.

1. Preservation and reperfusion injuries in liver allografts. An overview and synthesis of current studies;Clavien;Transplantation,1992

2. Validation of a current definition of early allograft dysfunction in liver transplant recipients and analysis of risk factors;Olthoff;Liver Transpl,2010

3. Risk factors for primary dysfunction after liver transplantation-a multivariate analysis;Ploeg;Transplantation,1993

4. Impact of donor graft steatosis on overall outcome and viral recurrence after liver transplantation for hepatitis C virus cirrhosis;Briceño;Liver Transpl,2009

5. Neutrophil and Kupffer cell-induced oxidant stress and ischemia-reperfusion injury in rat liver;Jaeschke;Am J Physiol,1991

Cited by 23 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Research progress on the role and inhibitors of Keap1 signaling pathway in inflammation;International Immunopharmacology;2024-11

2. Albumin promoter-driven FlpO expression induces efficient genetic recombination in mouse liver;American Journal of Physiology-Gastrointestinal and Liver Physiology;2024-05-01

3. Targeting PI3K/p‐Akt/eNOS, Nrf2/HO‐1, and NF‐κB/p53 signaling pathways by angiotensin 1–7 protects against liver injury induced by ischemia–reperfusion in rats;Cell Biochemistry and Function;2024-01

4. Activation of Nrf2 in fibroblasts promotes a skin aging phenotype via an Nrf2-miRNA-collagen axis;Matrix Biology;2022-11

5. p21‐activated kinase 4 inhibition protects against liver ischemia/reperfusion injury: Role of nuclear factor erythroid 2‐related factor 2 phosphorylation;Hepatology;2022-04-22