Pharmacological inhibition of TLR4/NF‐κB with TLR4‐IN‐C34 attenuated microcystin‐leucine arginine toxicity in bovine Sertoli cells
Author:
Affiliation:
1. Department of Animal Genetics, Breeding and Reproduction, College of Animal Science and TechnologyNortheast Agricultural University Harbin People's Republic of China
Funder
Ministry of Science and Technology of the People's Republic of China
Publisher
Wiley
Subject
Toxicology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/jat.3771
Reference62 articles.
1. Immunomodulatory Potency of Microcystin, an Important Water-Polluting Cyanobacterial Toxin
2. Microcystin-leucine arginine (MC-LR) induced inflammatory response in bovine sertoli cell via TLR4/NF-kB signaling pathway
3. Selenium (Na2SeO3) Upregulates Expression of Immune Genes and Blood–Testis Barrier Constituent Proteins of Bovine Sertoli Cell In Vitro
4. Sodium Selenite inhibits mitophagy, downregulation and mislocalization of blood-testis barrier proteins of bovine Sertoli cell exposed to microcystin-leucine arginine (MC-LR) via TLR4/NF-kB and mitochondrial signaling pathways blockage
5. TLR Signaling
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