Idiopathic pulmonary fibrosis fibroblasts become resistant to Fas ligand-dependent apoptosis via the alteration of decoy receptor 3-Reference-Cited by-同舟云学术

Idiopathic pulmonary fibrosis fibroblasts become resistant to Fas ligand-dependent apoptosis via the alteration of decoy receptor 3

Published:2016-07-14 Issue:1 Volume:240 Page:25-37
ISSN:0022-3417
Container-title:The Journal of Pathology
language:en
Short-container-title:J. Pathol.

Author:

Im Jintaek¹,Kim Kyutae²,Hergert Polla¹³,Nho Richard Seonghun¹

Affiliation:

1. Department of Medicine; University of Minnesota; Minneapolis MN USA

2. College of Biological Science; University of Minnesota; St. Paul MN USA

3. Lung Morphology Research Core Department; University of Minnesota; Minneapolis MN USA

Funder

National Heart, Lung and Blood Institute

Publisher

Wiley

Subject

Pathology and Forensic Medicine

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3. Idiopathic pulmonary fibrosis: current concepts;Ryu;Mayo Clin Proc,1998

4. Pathological alteration of FoxO3a activity promotes idiopathic pulmonary fibrosis fibroblast proliferation on type I collagen matrix;Nho;Am J Pathol,2011

5. FoxO3a (Forkhead Box O3a) deficiency protects idiopathic pulmonary fibrosis (IPF) fibroblasts from type I polymerized collagen matrix-induced apoptosis via caveolin-1 (cav-1) and Fas;Nho;PLoS One,2013

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