p116 Rip promotes myosin phosphatase activity in airway smooth muscle cells
Author:
Affiliation:
1. Department of Cellular and Molecular Biology The University of Texas Health Science Center at Tyler Tyler Texas
2. Department of Pathology and Laboratory Medicine University of British Columbia Vancouver BC Canada
Funder
Canadian Institutes of Health Research
Foundation for the National Institutes of Health
Publisher
Wiley
Subject
Cell Biology,Clinical Biochemistry,Physiology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcp.28949
Reference62 articles.
1. Regulation of smooth muscle contractile proteins by calmodulin and cyclic AMP;Adelstein R. S.;Federation Proceedings,1982
2. An inflammation-independent contraction mechanophenotype of airway smooth muscle in asthma
3. Smooth Muscle Specific Rac1 Deficiency Induces Hypertension by Preventing p116 RIP 3 ‐Dependent RhoA Inhibition
4. Airway smooth muscle relaxation results from a reduction in the frequency of Ca2+ oscillations induced by a cAMP-mediated inhibition of the IP3 receptor
5. Inhibition of Glycogen Synthase Kinase 3β Blocks Mesomesenchymal Transition and Attenuates Streptococcus pneumonia–Mediated Pleural Injury in Mice
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