<scp>MicroRNA</scp> ‐338‐3p as a therapeutic target in cardiac fibrosis through <scp>FGFR2</scp> suppression-Reference-Cited by-同舟云学术

MicroRNA ‐338‐3p as a therapeutic target in cardiac fibrosis through FGFR2 suppression

Published:2022-07-06 Issue:8 Volume:36 Page:
ISSN:0887-8013
Container-title:Journal of Clinical Laboratory Analysis
language:en
Short-container-title:Clinical Laboratory Analysis

Author:

Huang Cheng¹^ORCID,Wang Rui¹,Lu Jianjun²,He Yongli³,Wu Yueheng⁴,Ma Wuxia⁴,Xu Jindong⁵,Wu Zejia¹,Feng Zhe¹,Wu Min⁴^ORCID

Affiliation:

1. Department of Cardiology, Guangdong Provincial People's Hospital, Guangdong Academy of Medical Sciences Guangdong Cardiovascular Institute Guangzhou China

2. Department of Medical Services The First Affiliated Hopital of Sun Yat‐sen University Guangzhou China

3. Department of Intensive Care Unit Guangdong General Hospital Zhuhai Hospital Zhuhai Golden Bay Center Hospital Zhuhai China

4. Department of Cardiovascular Surgery, Guangdong Provincial People's Hospital, Guangdong Academy of Medical Sciences Guangdong Cardiovascular Institute Guangzhou China

5. Department of Anesthesiology Guangdong Provincial People's Hospital Guangzhou China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Microbiology (medical),Biochemistry (medical),Medical Laboratory Technology,Clinical Biochemistry,Public Health, Environmental and Occupational Health,Hematology,Immunology and Allergy

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcla.24584

Reference20 articles.

1. Cardiac fibrosis: new insights into the pathogenesis

2. The Predictive Role for ST2 in Patients with Acute Coronary Syndromes and Heart Failure

3. Dynamic Chromatin Targeting of BRD4 Stimulates Cardiac Fibroblast Activation

4. Targeting cardiac fibrosis with engineered T cells

5. Cardiac fibroblasts are essential for the adaptive response of the murine heart to pressure overload

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