Advanced glycation end-products induce skeletal muscle atrophy and dysfunction in diabetic mice via a RAGE-mediated, AMPK-down-regulated, Akt pathway

Author:

Chiu Chen-Yuan1,Yang Rong-Sen2,Sheu Meei-Ling3,Chan Ding-Cheng4,Yang Ting-Hua5,Tsai Keh-Sung6,Chiang Chih-Kang17,Liu Shing-Hwa189

Affiliation:

1. Institute of Toxicology, College of Medicine; National Taiwan University; Taipei Taiwan

2. Department of Orthopaedics, College of Medicine; National Taiwan University; Taipei Taiwan

3. Institute of Biomedical Sciences; National Chung Hsing University; Taichung Taiwan

4. Department of Geriatrics and Gerontology, College of Medicine; National Taiwan University; Taipei Taiwan

5. Department of Otolaryngology; National Taiwan University Hospital; Taipei Taiwan

6. Department of Laboratory Medicine, College of Medicine; National Taiwan University; Taipei Taiwan

7. Departments of Integrated Diagnostics and Therapeutics and Internal Medicine, College of Medicine and Hospital; National Taiwan University; Taipei Taiwan

8. Department of Paediatrics; National Taiwan University Hospital; Taipei Taiwan

9. Department of Medical Research; China Medical University Hospital, China Medical University; Taichung Taiwan

Funder

National Science Council

Publisher

Wiley

Subject

Pathology and Forensic Medicine

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