Podocyte is the major culprit accounting for the progression of chronic renal disease
Author:
Publisher
Wiley
Subject
Medical Laboratory Technology,Instrumentation,Histology,Anatomy
Reference45 articles.
1. In progressive nephropathies, overload of tubular cells with filtered proteins translates glomerular permeability dysfunction into cellular signals of interstitial inflammation.
2. Antiproteinuric Therapy while Preventing the Abnormal Protein Traffic in Proximal Tubule Abrogates Protein- and Complement-Dependent Interstitial Inflammation in Experimental Renal Disease
3. 1995. The role of nephron mass and of intraglomerular pressure in initiation and progression of experimental hypertensive-renal disorders. In: Hypertension: pathophysiology, diagnosis and management. editors. New York: Raven Press pp 1553-1588.
4. Combined effects of an angiotensin converting enzyme inhibitor and a calcium antagonist on renal injury
5. The Dysregulated Podocyte Phenotype
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