<i>Transferrin receptor 2 (Tfr2)</i> genetic deletion makes transfusion‐independent a murine model of transfusion‐dependent β‐thalassemia-Reference-Cited by-同舟云学术

Transferrin receptor 2 (Tfr2) genetic deletion makes transfusion‐independent a murine model of transfusion‐dependent β‐thalassemia

Published:2022-08-10 Issue:10 Volume:97 Page:1324-1336
ISSN:0361-8609
Container-title:American Journal of Hematology
language:en
Short-container-title:American J Hematol

Author:

Di Modica Simona Maria¹,Tanzi Emanuele¹,Olivari Violante¹²,Lidonnici Maria Rosa³,Pettinato Mariateresa¹²,Pagani Alessia¹,Tiboni Francesca³,Furiosi Valeria¹,Silvestri Laura¹²^ORCID,Ferrari Giuliana²³,Rivella Stefano⁴,Nai Antonella¹²^ORCID

Affiliation:

1. Regulation of Iron Metabolism Unit, Division of Genetics and Cell Biology Ospedale San Raffaele Milan Italy

2. Vita Salute San Raffaele University Milan Italy

3. San Raffaele Telethon Institute for Gene Therapy (SR‐TIGET) Ospedale San Raffaele Milan Italy

4. Division of Hematology, Department of Pediatrics Children's Hospital of Philadelphia Philadelphia Pennsylvania USA

Funder

American Society of Hematology

Cooley's Anemia Foundation

European Hematology Association

Fondazione Cariplo

Fondazione Telethon

Ministero della Salute

National Institute of Diabetes and Digestive and Kidney Diseases

Pennsylvania Department of Health

Publisher

Wiley

Subject

Hematology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/ajh.26673

Reference76 articles.

1. Integrated protein quality-control pathways regulate free α-globin in murine β-thalassemia

2. Heme-regulated eIF2α kinase in erythropoiesis and hemoglobinopathies

3. β-Thalassemias

4. Identification of erythroferrone as an erythroid regulator of iron metabolism

5. Erythroferrone contributes to hepcidin suppression and iron overload in a mouse model of β-thalassemia

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