Ca 2+ /calmodulin‐dependent protein kinase II equally induces sarcoplasmic reticulum Ca 2+ leak in human ischaemic and dilated cardiomyopathy-Reference-Cited by-同舟云学术

Ca 2+ /calmodulin‐dependent protein kinase II equally induces sarcoplasmic reticulum Ca 2+ leak in human ischaemic and dilated cardiomyopathy

Published:2014-09-08 Issue:12 Volume:16 Page:1292-1300
ISSN:1388-9842
Container-title:European Journal of Heart Failure
language:en
Short-container-title:Eur J Heart Fail

Author:

Fischer Thomas H.¹,Eiringhaus Jörg¹,Dybkova Nataliya¹,Förster Anna¹,Herting Jonas¹,Kleinwächter Astrid¹,Ljubojevic Senka²,Schmitto Jan D.³,Streckfuß‐Bömeke Katrin¹,Renner André⁴,Gummert Jan⁴,Hasenfuss Gerd¹⁵,Maier Lars S.⁶,Sossalla Samuel¹⁵

Affiliation:

1. Abteilung Kardiologie und Pneumologie/Herzzentrum Georg‐August‐Universität Göttingen Germany

2. Abteilung Kardiologie Medizinische Universitätsklinik Graz Austria

3. Abteilung Herz‐, Thorax‐, Gefäß‐ und Transplantationschirurgie Medizinische Hochschule Hannover Germany

4. Abteilung Thorax‐, Herz‐, Gefäßchirurgie Herz‐ und Diabeteszentrum Nordrheinwestfalen Bad Oeynhausen Germany

5. German Center for Cardiovascular Research (DZHK) Partner Site Goettingen Germany

6. Klinik und Poliklinik für Innere Medizin II Uiversitätsklinikum Regensburg Germany

Funder

Deutsche Forschungsgemeinschaft

Deutsche Stiftung fÃ¼r Herzforschung

Publisher

Wiley

Subject

Cardiology and Cardiovascular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/ejhf.163

Reference23 articles.

1. Identification and Expression of δ-Isoforms of the Multifunctional Ca 2+ /Calmodulin-Dependent Protein Kinase in Failing and Nonfailing Human Myocardium

2. Inhibition of Elevated Ca 2+ /Calmodulin-Dependent Protein Kinase II Improves Contractility in Human Failing Myocardium

3. Activity of cAMP-dependent protein kinase and Ca2+/calmodulin-dependent protein kinase in failing and nonfailing human hearts

4. The ryanodine receptor leak: how a tattered receptor plunges the failing heart into crisis

5. Transgenic CaMKIIδ C Overexpression Uniquely Alters Cardiac Myocyte Ca 2+ Handling

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