CRISPR-Cas9 base editing of pathogenic CaMKIIδ improves cardiac function in a humanized mouse model-Reference-Cited by-同舟云学术

CRISPR-Cas9 base editing of pathogenic CaMKIIδ improves cardiac function in a humanized mouse model

Published:2024-01-02 Issue:1 Volume:134 Page:
ISSN:1558-8238
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:

Author:

Lebek Simon^ORCID,Caravia Xurde M.^ORCID,Straub Leon G.^ORCID,Alzhanov Damir,Tan Wei,Li Hui^ORCID,McAnally John R.,Chen Kenian,Xu Lin^ORCID,Scherer Philipp E.^ORCID,Liu Ning^ORCID,Bassel-Duby Rhonda^ORCID,Olson Eric N.

Funder

National Institutes of Health

Fondation Leducq

British Heart Foundation

German Research Foundation

American Heart Association

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Link

https://www.jci.org/articles/view/175164/files/pdf

Reference46 articles.

1. Heart Disease and Stroke Statistics—2021 Update

2. Enhanced CaMKII-Dependent Late I Na Induces Atrial Proarrhythmic Activity in Patients With Sleep-Disordered Breathing

3. Diabetes increases mortality after myocardial infarction by oxidizing CaMKII

4. Challenges and Opportunities for Therapeutic Targeting of Calmodulin Kinase II in Heart

5. CaMKII inhibitors: from research tools to therapeutic agents

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