<scp>LINC00365</scp> inhibited lung adenocarcinoma progression and glycolysis via sponging <scp>miR</scp> ‐429/ <scp>KCTD12</scp> axis-Reference-Cited by-同舟云学术

LINC00365 inhibited lung adenocarcinoma progression and glycolysis via sponging miR ‐429/ KCTD12 axis

Published:2022-04-15 Issue:8 Volume:37 Page:1853-1866
ISSN:1520-4081
Container-title:Environmental Toxicology
language:en
Short-container-title:Environmental Toxicology

Author:

Zhang Cheng‐Wei¹,Zhou Bin¹,Liu Yan‐Chao¹,Su Li‐Wei¹,Meng Jie¹,Li Shao‐Lei²,Wang Xue‐Long¹^ORCID

Affiliation:

1. Department of Thoracic Surgery Capital Medical University Electric Power Teaching Hospital Beijing China

2. Department of Thoracic Surgery II, Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education) Peking University Cancer Hospital and Institute Beijing China

Publisher

Wiley

Subject

Health, Toxicology and Mutagenesis,Management, Monitoring, Policy and Law,Toxicology,General Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/tox.23532

Reference53 articles.

1. SMARCA4 mutations in KRAS ‐mutant lung adenocarcinoma: a multi‐cohort analysis

2. Treatment Sequencing for Anaplastic Lymphoma Kinase-Rearranged Non-Small-Cell Lung Cancer

3. The expression of trefoil factor 3 is related to histologic subtypes and invasiveness in lung adenocarcinoma

4. RNF115-mediated ubiquitination of p53 regulates lung adenocarcinoma proliferation

5. MiR‐423‐5p aggravates lung adenocarcinoma via targeting CADM1

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