Immunopharmacological effect of β‐ d ‐mannuronic acid (M2000), as a new immunosuppressive drug, on gene expression of miR‐155 and its target molecules (SOCS1, SHIP1) in a clinical trial on rheumatoid arthritis patients-Reference-Cited by-同舟云学术

Immunopharmacological effect of β‐ d ‐mannuronic acid (M2000), as a new immunosuppressive drug, on gene expression of miR‐155 and its target molecules (SOCS1, SHIP1) in a clinical trial on rheumatoid arthritis patients

Published:2019-11 Issue:3 Volume:81 Page:295-304
ISSN:0272-4391
Container-title:Drug Development Research
language:en
Short-container-title:Drug Dev Res

Author:

Mortazavi‐Jahromi Seyed Shahabeddin¹^ORCID,Aslani Mona¹,Omidian Saiedeh¹,Ahmadzadeh Arman²^ORCID,Rezaieyazdi Zahra³^ORCID,Mirshafiey Abbas¹^ORCID

Affiliation:

1. Department of Immunology, School of Public HealthTehran University of Medical Sciences Tehran Iran

2. Department of Rheumatology, Loghman Hakim HospitalShahid Beheshti University of Medical Sciences Tehran Iran

3. Rheumatic Diseases Research CenterMashhad University of Medical Sciences Mashhad Iran

Publisher

Wiley

Subject

Drug Discovery

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/ddr.21619

Reference58 articles.

1. A phase I/II randomized, controlled, clinical trial for assessment of the efficacy and safety of β-d-mannuronic acid in rheumatoid arthritis patients

2. MicroRNA-155—at the Critical Interface of Innate and Adaptive Immunity in Arthritis

3. Src homology 2 domain-containing inositol-5-phosphatase 1 (SHIP1) negatively regulates TLR4-mediated LPS response primarily through a phosphatase activity- and PI-3K-independent mechanism

4. Role of SHIP1 in Invariant NKT Cell Development and Functions

5. Effects of β-D-mannuronic acid, as a novel non-steroidal anti-inflammatory medication within immunosuppressive properties, on IL17, RORγt, IL4 and GATA3 gene expressions in rheumatoid arthritis patients

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