Epidermal growth factor receptor (EGFR)-MAPK-nuclear factor(NF)-κB-IL8: A possible mechanism of particulate matter(PM) 2.5-induced lung toxicity

Author:

Jeong Seung-Chan12,Cho Yoon1,Song Mi-Kyung3,Lee Eunil2,Ryu Jae-Chun14

Affiliation:

1. Cellular and Molecular Toxicology Laboratory; Korea Institute of Science & Technology; P.O. Box 131, Cheongryang Seoul 130-650 Korea

2. Department of Preventive Medicine; Korea University, Korea Project for Reducing Light Pollution Effects to Human and Ecosystem; Korea

3. National Center for Efficacy evaluation for Respiratory disease product, Jeonbuk Department of Inhalation Research; Korea Institute of Toxicology; 30 Baehak1-gil Jeongeup Jeollabuk-do 53212 Republic of Korea

4. Department of Pharmacology and Toxicology, Human and Environmental Toxicology; Korea University of Science and Technology; Gajeong-Ro 217, Yuseong-gu Daejeon 305-350 Korea

Funder

The Ecoinnovation Project

Korea Institute of Science and Technology

Publisher

Wiley

Subject

Health, Toxicology and Mutagenesis,Management, Monitoring, Policy and Law,Toxicology,General Medicine

Reference50 articles.

1. Health risk by inhalation of PM2.5 in the metropolitan zone of the City of Mexico;Diaz;Ecotoxicol Environ Safety.,2009

2. PM10 and PM2.5 and health risk assessment for heavy metals in a typical factory for cathode ray tube television recycling;Fang;Environ Sci Technol.,2013

3. Effect of PM2.5 collected during the dust and non-dust periods on the viability and gap junctional intercellular communication in human lung fibroblasts;Wang;J Hygiene Res.,2006

4. PM2.5, PM10 and health risk assessment of heavy metals in a typical printed circuit noards manufacturing workshop;Zhou;J Environ Sci.,1993

5. Extraction method for manipulation of water- and organic-soluble extracts of PM2.5 in Korean winter season and its chemical composition;Shin;Toxicol Environ Health Sci.,2013

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