Facilitation of Ca2+ -induced opening of the mitochondrial permeability transition pore either by nicotinamide nucleotide transhydrogenase deficiency or statins treatment
Author:
Affiliation:
1. Faculdade de Ciências Médicas; Departamento de Patologia Clínica, UNICAMP; Rua Cinco de Junho, 350 CEP 13083-877 Campinas SP Brasil
2. Instituto de Biologia; Departamento de Biologia Estrutural e Funcional, UNICAMP; SP Brasil
Funder
Coordenação de Aperfeiçoamento de Pessoal de Nível Superior
Fundação de Amparo à Pesquisa do Estado de São Paulo
Conselho Nacional de Desenvolvimento Científico e Tecnológico
Publisher
Wiley
Subject
Cell Biology,General Medicine
Reference21 articles.
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2. Protective effects of l-carnitine and piracetam against mitochondrial permeability transition and PC3 cell necrosis induced by simvastatin;Costa;Eur J Pharmacol,2013
3. Mitochondria as a source of reactive oxygen and nitrogen species: From molecular mechanisms to human health;Figueira;Antioxid Redox Signal,2013
4. A direct comparison of metabolic responses to high fat diet in C57BL/6J and C57BL/6NJ mice;Fisher-Wellman;Diabetes,2016
5. Impact of a novel homozygous mutation in nicotinamide nucleotide transhydrogenase on mitochondrial DNA integrity in a case of familial glucocorticoid deficiency;Fujisawa;BBA Clinical,2015
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