Mitochondria as a Source of Reactive Oxygen and Nitrogen Species: From Molecular Mechanisms to Human Health-Reference-Cited by-同舟云学术

Mitochondria as a Source of Reactive Oxygen and Nitrogen Species: From Molecular Mechanisms to Human Health

Published:2013-06 Issue:16 Volume:18 Page:2029-2074
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Figueira Tiago R.¹,Barros Mario H.²,Camargo Anamaria A.³⁴,Castilho Roger F.¹,Ferreira Julio C.B.⁵,Kowaltowski Alicia J.⁶,Sluse Francis E.⁷,Souza-Pinto Nadja C.⁶,Vercesi Anibal E.¹

Affiliation:

1. Department of Clinical Pathology, Faculty of Medical Sciences, State University of Campinas, Campinas, Brazil.

2. Department of Microbiology, Institute of Biomedical Sciences, University of São Paulo, São Paulo, Brazil.

3. Ludwig Institute for Cancer Research, São Paulo, Brazil.

4. Molecular Oncology Center, Sirio-Libanese Hospital, São Paulo, Brazil.

5. Department of Anatomy, Institute of Biomedical Sciences, University of São Paulo, São Paulo, Brazil.

6. Departamento de Bioquímica, Instituto de Química, Universidade de São Paulo, São Paulo, Brazil.

7. Laboratory of Bioenergetics, Department of Life Sciences, Faculty of Sciences, University of Liege, Liege, Belgium.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2012.4729

Reference568 articles.

1. Respiratory Complex III Is Required to Maintain Complex I in Mammalian Mitochondria

2. Regulation of intermediary metabolism by the PKCδ signalosome in mitochondria

3. Upregulation of Nox4 by Hypertrophic Stimuli Promotes Apoptosis and Mitochondrial Dysfunction in Cardiac Myocytes

4. Kinetic model of the inhibition of respiration by endogenous nitric oxide in intact cells

5. A novel mutation in the gene encoding TIMM8a, a component of the mitochondrial protein translocase complexes, in a Spanish familial case of deafness-dystonia (Mohr–Tranebjaerg) syndrome

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