Palmitic acid methyl ester induces cardiac hypertrophy through activating the GPR receptor‐mediated changes of intracellular calcium concentrations and mitochondrial functions-Reference-Cited by-同舟云学术

Palmitic acid methyl ester induces cardiac hypertrophy through activating the GPR receptor‐mediated changes of intracellular calcium concentrations and mitochondrial functions

Published:2022-12-20 Issue:1 Volume:238 Page:242-256
ISSN:0021-9541
Container-title:Journal of Cellular Physiology
language:en
Short-container-title:Journal Cellular Physiology

Author:

Lien Chih‐Feng¹²,Chiu Hung‐Wen³,Lee Wen‐Sen⁴^ORCID,Lin Jian‐Hong⁵,Wang Yi‐Shun⁶⁷,Ting Pei‐Ching⁸,Luo Yu‐Po⁸,Chang Jui‐Chih⁸⁹,Yang Kun‐Ta³¹⁰^ORCID

Affiliation:

1. Institute of Medical Sciences, College of Medicine Tzu Chi University Hualien Taiwan

2. Department of Medicine, Division of Cardiology, Tri‐Service General Hospital National Defense Medical Center Taipei Taiwan

3. Master Program in Physiological and Anatomical, Medicine School of Medicine Tzu Chi University Hualien Taiwan

4. Graduate Institute of Medical Sciences, College of Medicine Taipei Medical University Taipei Taiwan

5. Department of Surgery, Division of Experimental Surgery, Hualien Tzu Chi Hospital Buddhist Tzu Chi Medical Foundation Hualien Taiwan

6. Department of Life Science Tzu Chi University Hualien Taiwan

7. Master Program in Biomedical Science, School of Medicine Tzu Chi University Hualien Taiwan

8. Department of Surgery, Hualien Tzu Chi Hospital Buddhist Tzu Chi Medical Foundation Hualien Taiwan

9. Department of Surgery, School of Medicine Tzu Chi University Hualien Taiwan

10. Department of Physiology, School of Medicine, College of Medicine Tzu Chi University Hualien Taiwan

Funder

Tzu Chi University

Ministry of Science and Technology, Taiwan

Publisher

Wiley

Subject

Cell Biology,Clinical Biochemistry,Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcp.30922

Reference40 articles.

1. Transverse aortic constriction leads to accelerated heart failure in mice lacking PPAR- coactivator 1

2. Mechanism of modulation of the voltage-gated skeletal and cardiac muscle sodium channels by fatty acids

3. Calcium, ATP, and ROS: a mitochondrial love-hate triangle

4. Intermittent Hypoxia Inhibits Na+-H+ Exchange-Mediated Acid Extrusion Via Intracellular Na+ Accumulation in Cardiomyocytes

5. Intermittent Hypoxia Prevents Myocardial Mitochondrial Ca2+ Overload and Cell Death during Ischemia/Reperfusion: The Role of Reactive Oxygen Species

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