Downregulation of PTEN by sodium orthovanadate protects the myocardium against ischemia/reperfusion injury after chronic atorvastatin treatment
Author:
Affiliation:
1. Department of Cardiology Beijing Anzhen Hospital, Capital Medical University Beijing China
2. Children’s Hospital, Capital Medical University, National Center for Children’s Health Beijing China
Funder
National Natural Science Foundation of China
Publisher
Wiley
Subject
Cell Biology,Molecular Biology,Biochemistry
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcb.27651
Reference21 articles.
1. Pretreatment With Simvastatin Attenuates Myocardial Dysfunction After Ischemia and Chronic Reperfusion
2. Atorvastatin, administered at the onset of reperfusion, and independent oflipid lowering, protects the myocardiumby up-regulating a pro-survival pathway
3. Induction of RISK by HMG-CoA reductase inhibition affords cardioprotection after myocardial infarction
4. Failure to protect the myocardium against ischemia/reperfusion injury after chronic atorvastatin treatment is recaptured by acute atorvastatin treatment
5. The role of phosphoinositide-3 kinase and PTEN in cardiovascular physiology and disease
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3. MicroRNA-29b reduces myocardial ischemia–reperfusion injury in rats via down-regulating PTEN and activating the Akt/eNOS signaling pathway;Journal of Thrombosis and Thrombolysis;2021-08-09
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