Motheaten (me/me) mice deficient in SHP-1 are less susceptible to focal cerebral ischemia
Author:
Publisher
Wiley
Subject
Cellular and Molecular Neuroscience
Reference44 articles.
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2. The major SHP-1-binding, tyrosine-phosphorylated protein in macrophages is a member of the KIR/LIR family and an SHP-1 substrate
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4. T Cell Developmental Defects in ‘Viable Motheaten’ Mice Deficient in SHP-1 Protein-Tyrosine Phosphatase. Developmental Defects are Corrected in vitro in the Presence of Normal Hematopoietic-Origin Stromal Cells and in vivo by Exogenous IL-7
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