T Cell Developmental Defects in ‘Viable Motheaten’ Mice Deficient in SHP-1 Protein-Tyrosine Phosphatase. Developmental Defects are Corrected in vitro in the Presence of Normal Hematopoietic-Origin Stromal Cells and in vivo by Exogenous IL-7-Reference-Cited by-同舟云学术

T Cell Developmental Defects in ‘Viable Motheaten’ Mice Deficient in SHP-1 Protein-Tyrosine Phosphatase. Developmental Defects are Corrected in vitro in the Presence of Normal Hematopoietic-Origin Stromal Cells and in vivo by Exogenous IL-7

Published:2002-03 Issue:2 Volume:18 Page:119-130
ISSN:0896-8411
Container-title:Journal of Autoimmunity
language:en
Short-container-title:Journal of Autoimmunity

Author:

Christianson Sherri W,Greiner Dale L,Deluca Dominick,Leif Jean,Phillips Nancy E,Hayes Sandra M,Hayashi Shin-Ichi,Joliat Melissa J,Lyons Bonnie L,Shultz Leonard D

Publisher

Elsevier BV

Subject

Immunology,Immunology and Allergy

Reference53 articles.

1. Mutations at the murine motheaten locus are within the hematopoietic cell protein-tyrosine phosphatase (Hcph) gene;Shultz;Cell,1993

2. Motheaten and viable motheaten mice have mutations in the haematopoietic cell phosphatase gene;Tsui;Nat. Genet.,1993

3. Severe defects in immunity and hematopoiesis caused by SHP-1 protein-tyrosine-phosphatase deficiency;Shultz;Trends Biotechnol.,1997

4. The role of phosphotyrosine phosphatases in haematopoietic cell signal transduction;Frearson;Bioessays,1997

5. Protein tyrosine phosphatases in signal transduction;Neel;Curr. Opin. Cell Biol.,1997

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