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EGFR and MEK Blockade in Triple Negative Breast Cancer Cells

Published:2015-10-08 Issue:12 Volume:116 Page:2778-2785
ISSN:0730-2312
Container-title:Journal of Cellular Biochemistry
language:en
Short-container-title:J. Cell. Biochem.

Author:

Maiello Monica Rosaria¹,D'Alessio Amelia¹,Bevilacqua Simona¹,Gallo Marianna¹,Normanno Nicola¹,De Luca Antonella¹

Affiliation:

1. Cell Biology and Biotherapy Unit; Istituto Nazionale Tumori “Fondazione G. Pascale”-IRCCS; Naples Italy

Funder

Associazione Italiana per la Ricerca sul Cancro

Publisher

Wiley

Subject

Cell Biology,Molecular Biology,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/jcb.25220/fullpdf

Reference20 articles.

1. Inhibition of proliferation and induction of apoptosis in breast cancer cells by the epidermal growth factor receptor (EGFR) tyrosine kinase inhibitor ZD1839 ('Iressa') is independent of EGFR expression level;Campiglio;J Cell Physiol,2004

2. AKT inhibition relieves feedback suppression of receptor tyrosine kinase expression and activity;Chandarlapaty;Cancer Cell,2011

3. Quantitative analysis of dose-effect relationships: the combined effects of multiple drugs or enzyme inhibitors;Chou;Adv Enzyme Regul,1984

4. Epidermal growth factor receptor as a potential therapeutic target in triple-negative breast cancer;Corkery;Ann Oncol,2009

5. Genome and transcriptome sequencing in prospective metastatic triple-negative breast cancer uncovers therapeutic vulnerabilities;Craig;Mol Cancer Ther,2013

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