Absence of DCIR1 reduces the mortality rate of endotoxemic hepatitis in mice

Author:

Ishiguro Toshifumi1,Fukawa Tetsuya1,Akaki Kotaro1,Nagaoka Koji1,Takeda Tatsuki1,Iwakura Yoichiro2,Inaba Kayo1,Takahara Kazuhiko1

Affiliation:

1. Department of Animal Development and Physiology; Graduate School of Biostudies; Kyoto University; Yoshida-Konoe, Sakyo Kyoto Japan

2. Research Institute for Biomedical Sciences; Tokyo University of Science; Noda Chiba Japan

Funder

Japan Society for the Promotion of Science

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Reference39 articles.

1. Signaling by myeloid C-type lectin receptors in immunity and homeostasis;Sancho;Annu. Rev. Immunol.,2011

2. Collaborative induction of inflammatory responses by dectin-1 and Toll-like receptor 2;Gantner;J. Exp. Med.,2003

3. Dectin-1 is an extracellular pathogen sensor for the induction and processing of IL-1β via a noncanonical caspase-8 inflammasome;Gringhuis;Nat. Immunol.,2012

4. Signalling through C-type lectin receptors: shaping immune responses;Geijtenbeek;Nat. Rev. Immunol.,2009

5. APCs express DCIR, a novel C-type lectin surface receptor containing an immunoreceptor tyrosine-based inhibitory motif;Bates;J. Immunol.,1999

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