UHRF1 downmodulation enhances antitumor effects of histone deacetylase inhibitors in retinoblastoma by augmenting oxidative stress‐mediated apoptosis-Reference-Cited by-同舟云学术

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UHRF1 downmodulation enhances antitumor effects of histone deacetylase inhibitors in retinoblastoma by augmenting oxidative stress‐mediated apoptosis

Published:2019-12-13 Issue:2 Volume:14 Page:329-346
ISSN:1574-7891
Container-title:Molecular Oncology
language:en
Short-container-title:Mol Oncol

Author:

Kim Jong Kyong¹^ORCID,Kan Guangyan¹,Mao Yu¹,Wu Zhixuan¹,Tan Xionghong¹^ORCID,He Heng¹,Lee Chunsik¹

Affiliation:

1. State Key Laboratory of Ophthalmology Zhongshan Ophthalmic Center Sun Yat‐sen University Guangzhou China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Cancer Research,Genetics,Molecular Medicine,General Medicine,Oncology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/1878-0261.12607

Reference41 articles.

1. Chromatin remodelers HELLS and UHRF1 mediate the epigenetic deregulation of genes that drive retinoblastoma tumor progression

2. Differentiation therapy for the treatment of t(8;21) acute myeloid leukemia using histone deacetylase inhibitors

3. Increasing role of UHRF1 in the reading and inheritance of the epigenetic code as well as in tumorogenesis

4. Inhibition of histone deacetylases in cancer therapy: lessons from leukaemia

5. Chemotherapy for Retinoblastoma

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