Activation of protein phosphatase 1 by a selective phosphatase disrupting peptide reduces sarcoplasmic reticulum Ca 2+ leak in human heart failure-Reference-Cited by-同舟云学术

Activation of protein phosphatase 1 by a selective phosphatase disrupting peptide reduces sarcoplasmic reticulum Ca 2+ leak in human heart failure

Published:2018-09-07 Issue:12 Volume:20 Page:1673-1685
ISSN:1388-9842
Container-title:European Journal of Heart Failure
language:en
Short-container-title:Eur J Heart Fail

Author:

Fischer Thomas H.¹²³,Eiringhaus Jörg¹³,Dybkova Nataliya¹³,Saadatmand Alireza⁴,Pabel Steffen³⁵,Weber Silvio⁶,Wang Yansong⁷,Köhn Maja⁷⁸,Tirilomis Theodor⁹,Ljubojevic Senka¹⁰,Renner André¹¹,Gummert Jan¹¹,Maier Lars S.⁵,Hasenfuß Gerd¹³,El‐Armouche Ali⁶,Sossalla Samuel¹³⁵

Affiliation:

1. Klinik für Kardiologie und PneumologieGeorg‐August‐Universität Göttingen Germany

2. Medizinische Klinik II, Kardiologie, Angiologie, Pneumologie, Klinikum Coburg Germany

3. Deutsches Zentrum für Herz‐Kreislauf Forschung (DZHK), Standort Göttingen Germany

4. Abt. Molekulare Kardiologie und EpigenetikUniversitätsklinikum Heidelberg Germany

5. Klinik und Poliklinik für Innere Medizin IIUniversitätsklinikum Regensburg Germany

6. Institut für PharmakologieTechnische Universität Dresden Germany

7. European Molecular Biology LaboratoryGenome Biology Unit Heidelberg Germany

8. Centre for Biological Signalling Studies (BIOSS) and Faculty of BiologyUniversity of Freiburg Germany

9. Klinik für Thorax‐, Herz‐, GefäßchirurgieGeorg‐August‐Universität Göttingen Germany

10. Abteilung für KardiologieMedizinische Universität Graz Austria

11. Abteilung für Herz‐ und Transplantationschirurgie, Herz‐ und Diabeteszentrum Bad Oeynhausen Germany

Publisher

Wiley

Subject

Cardiology and Cardiovascular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/ejhf.1297

Reference33 articles.

1. The ryanodine receptor provides high throughput Ca2+-release but is precisely regulated by networks of associated proteins: a focus on proteins relevant to phosphorylation

2. Transgenic CaMKIIδ C Overexpression Uniquely Alters Cardiac Myocyte Ca 2+ Handling

3. Ca 2+ /Calmodulin-Dependent Protein Kinase II and Protein Kinase A Differentially Regulate Sarcoplasmic Reticulum Ca 2+ Leak in Human Cardiac Pathology

4. Calcium/Calmodulin-Dependent Protein Kinase II Contributes to Cardiac Arrhythmogenesis in Heart Failure

5. Ca 2+ /calmodulin‐dependent protein kinase II equally induces sarcoplasmic reticulum Ca 2+ leak in human ischaemic and dilated cardiomyopathy

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