Hepatoprotective action of schisandrin B against carbon tetrachloride toxicity was mediated by both enhancement of mitochondrial glutathione status and induction of heat shock proteins in mice-Reference-Cited by-同舟云学术

Hepatoprotective action of schisandrin B against carbon tetrachloride toxicity was mediated by both enhancement of mitochondrial glutathione status and induction of heat shock proteins in mice

Published:2003 Issue:1-2 Volume:19 Page:33-42
ISSN:0951-6433
Container-title:BioFactors
language:en
Short-container-title:BioFactors

Author:

Tang Man Ho,Chiu Po Yee,Ko Kam Ming

Publisher

Wiley

Subject

Clinical Biochemistry,Molecular Medicine,General Medicine,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/biof.5520190105/fullpdf

Reference24 articles.

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2. P.Y. Chiu and K.M. Ko, Time dependent enhancement in mitochondrial glutathione status and ATP generation capacity by schisandrin B treatment decreases the susceptibility of rat hearts to ischemia-reperfusion injury, Biofactors (in press).

3. Stable overexpression of the constitutive form of heat shock protein 70 confers oxidative protection;Chong;J. Mol. Cell Cardiol.,1998

4. A subfamily of stress proteins facilitates translocation of secretory and mitochondrial precursor polypeptides;Deshaies;Nature,1988

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