Bioflavonoids promote stable translocations betweenMLL-AF9breakpoint cluster regions independent of normal chromosomal context: Model system to screen environmental risks-Reference-Cited by-同舟云学术

Bioflavonoids promote stable translocations betweenMLL-AF9breakpoint cluster regions independent of normal chromosomal context: Model system to screen environmental risks

Published:2018-11-02 Issue:2 Volume:60 Page:154-167
ISSN:0893-6692
Container-title:Environmental and Molecular Mutagenesis
language:en
Short-container-title:Environ Mol Mutagen

Author:

Bariar Bhawana¹,Vestal C. Greer¹,Deem Bradley¹,Goodenow Donna¹,Ughetta Mimi¹,Engledove R. Warren¹,Sahyouni Mark¹,Richardson Christine¹

Affiliation:

1. Department of Biological Sciences; University of North Carolina at Charlotte; 9201 University City Boulevard, Charlotte North Carolina

Funder

National Institutes of Health

NIH/NCI

Publisher

Wiley

Subject

Health, Toxicology and Mutagenesis,Genetics(clinical),Epidemiology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/em.22245/fullpdf

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3. Bioflavonoids as poisons of human topoisomerase IIalpha and IIbeta;Bandele;Biochemistry,2007

4. Long-term effects of chromatin remodeling and DNA damage in stem cells induced by environmental and dietary agents;Bariar;J Environ Pathol Toxicol Oncol,2013

5. Dietary flavonoids induce MLL translocations in primary human CD34+ cells;Barjesteh van Waalwijk van Doorn-Khosrovani;Carcinogenesis,2007

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