Induction of autophagic cell death in human ovarian carcinoma cells by Antrodia salmonea through increased reactive oxygen species generation-Reference-Cited by-同舟云学术

Induction of autophagic cell death in human ovarian carcinoma cells by Antrodia salmonea through increased reactive oxygen species generation

Published:2018-12-24 Issue:7 Volume:234 Page:10747-10760
ISSN:0021-9541
Container-title:Journal of Cellular Physiology
language:en
Short-container-title:J Cell Physiol

Author:

Yang Hsin‐Ling¹^ORCID,Lin Ruei‐Wan¹,Karuppaiya Palaniyandi¹,Mathew Dony Chacko²^ORCID,Way Tzong‐Der³,Lin Hui‐Chang⁴,Lee Chuan‐Chen⁵,Hseu You‐Cheng²⁵⁶⁷^ORCID

Affiliation:

1. Institute of Nutrition, College of Biopharmaceutical and Food Sciences China Medical University Taichung Taiwan

2. Department of Cosmeceutics College of Biopharmaceutical and Food Sciences, China Medical University Taichung Taiwan

3. Department of Life Sciences China Medical University Taichung Taiwan

4. School of Pharmacy China Medical University Taichung Taiwan

5. Department of Health and Nutrition Biotechnology Asia University Taichung Taiwan

6. Chinese Medicine Research Center China Medical University Taichung Taiwan

7. Research Center of Chinese Herbal Medicine China Medical University Taichung Taiwan

Funder

Ministry of Science and Technology, Taiwan

Publisher

Wiley

Subject

Cell Biology,Clinical Biochemistry,Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcp.27749

Reference55 articles.

1. Response of Estrogen Receptor-Positive Breast Cancer Tumorspheres to Antiestrogen Treatments

2. Lysosomal membrane permeabilization in cell death

3. Antrodia salmonea induces G2 cell-cycle arrest in human triple-negative breast cancer (MDA-MB-231) cells and suppresses tumor growth in athymic nude mice

4. Inhibition of ROS production, autophagy or apoptosis signaling reversed the anticancer properties of Antrodia salmonea in triple-negative breast cancer (MDA-MB-231) cells

5. Simultaneous activation and inhibition of autophagy sensitizes cancer cells to chemotherapy

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