Androgen receptor splice variants contribute to prostate cancer aggressiveness through induction of EMT and expression of stem cell marker genes
Author:
Affiliation:
1. Department of Pathology; Karmanos Cancer Institute; Wayne State University School of Medicine; Detroit Michigan
2. Department of Oncology; Karmanos Cancer Institute; Wayne State University School of Medicine; Detroit Michigan
Funder
National Institutes of Health
Publisher
Wiley
Subject
Urology,Oncology
Reference37 articles.
1. A luminal epithelial stem cell that is a cell of origin for prostate cancer;Wang;Nature,2009
2. AR intragenic deletions linked to androgen receptor splice variant expression and activity in models of prostate cancer progression;Li;Oncogene,2012
3. Androgen receptor variants occur frequently in castration resistant prostate cancer metastases;Zhang;PLoS ONE,2011
4. Expression of androgen receptor splice variants in prostate cancer bone metastases is associated with castration-resistance and short survival;Hornberg;PLoS ONE,2011
5. Constitutively active androgen receptor splice variants expressed in castration-resistant prostate cancer require full-length androgen receptor;Watson;Proc Natl Acad Sci USA,2010
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