AR intragenic deletions linked to androgen receptor splice variant expression and activity in models of prostate cancer progression-Reference-Cited by-同舟云学术

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AR intragenic deletions linked to androgen receptor splice variant expression and activity in models of prostate cancer progression

Published:2012-01-23 Issue:45 Volume:31 Page:4759-4767
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Li Y,Hwang T H,Oseth L A,Hauge A,Vessella R L,Schmechel S C,Hirsch B,Beckman K B,Silverstein K A,Dehm S M

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

http://www.nature.com/articles/onc2011637.pdf

Reference42 articles.

1. Chen Y, Clegg NJ, Scher HI . Anti-androgens and androgen-depleting therapies in prostate cancer: new agents for an established target. Lancet Oncol 2009; 10: 981–991.

2. Heemers HV, Tindall DJ . Androgen receptor (AR) coregulators: a diversity of functions converging on and regulating the AR transcriptional complex. Endocr Rev 2007; 28: 778–808.

3. Scher HI, Sawyers CL . Biology of progressive, castration-resistant prostate cancer: directed therapies targeting the androgen-receptor signaling axis. J Clin Oncol 2005; 23: 8253–8261.

4. Taplin ME . Drug insight: role of the androgen receptor in the development and progression of prostate cancer. Nat Clin Pract Oncol 2007; 4: 236–244.

5. Attard G, Swennenhuis JF, Olmos D, Reid AH, Vickers E, A’Hern R et al. Characterization of ERG, AR and PTEN gene status in circulating tumor cells from patients with castration-resistant prostate cancer. Cancer Res 2009; 69: 2912–2918.

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