Lupus-Associated Functional Polymorphism inPNPCauses Cell Cycle Abnormalities and Interferon Pathway Activation in Human Immune Cells-Reference-Cited by-同舟云学术

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Lupus-Associated Functional Polymorphism inPNPCauses Cell Cycle Abnormalities and Interferon Pathway Activation in Human Immune Cells

Published:2017-11-09 Issue:12 Volume:69 Page:2328-2337
ISSN:2326-5191
Container-title:Arthritis & Rheumatology
language:en
Short-container-title:Arthritis Rheumatol

Author:

Ghodke-Puranik Yogita¹,Dorschner Jessica M.¹,Vsetecka Danielle M.¹,Amin Shreyasee²,Makol Ashima²,Ernste Floranne²,Osborn Thomas²,Moder Kevin²,Chowdhary Vaidehi²,Eliopoulos Elias³,Zervou Maria I.³,Goulielmos George N.³,Jensen Mark A.¹,Niewold Timothy B.¹

Affiliation:

1. New York University School of Medicine; New York New York

2. Mayo Clinic; Rochester Minnesota

3. Medical School of Crete; Heraklion Greece

Funder

Mayo Clinic

Colton Center for Autoimmunity

National Institutes of Health

Rheumatology Research Foundation

CureJM Foundation

Mayo Clinic Foundation

Lupus Foundation of Minnesota

Publisher

Wiley

Subject

Immunology,Rheumatology,Immunology and Allergy

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/art.40304/fullpdf

Reference41 articles.

1. Unraveling the genetics of systemic lupus erythematosus;Harley;Springer Semin Immunopathol,2006

2. Immune interferon in the circulation of patients with autoimmune disease;Hooks;N Engl J Med,1979

3. High serum IFN-α activity is a heritable risk factor for systemic lupus erythematosus;Niewold;Genes Immun,2007

4. Genetics of the type I interferon pathway in systemic lupus erythematosus;Ghodke-Puranik;Int J Clin Rheumatol,2013

5. Cutting edge: autoimmune disease risk variant of STAT4 confers increased sensitivity to IFN-α in lupus patients in vivo;Kariuki;J Immunol,2009

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