Sub‐chronic administration of benzo[a]pyrene disrupts hippocampal long‐term potentiation via inhibiting CaMK II / PKC / PKA‐ERK‐CREB signaling in rats-Reference-Cited by-同舟云学术

Sub‐chronic administration of benzo[a]pyrene disrupts hippocampal long‐term potentiation via inhibiting CaMK II / PKC / PKA‐ERK‐CREB signaling in rats

Published:2020-04-07 Issue:9 Volume:35 Page:961-970
ISSN:1520-4081
Container-title:Environmental Toxicology
language:en
Short-container-title:Environmental Toxicology

Author:

Lyu Yi¹²,Ren Xue‐Ke¹,Zhang Hui‐Fang¹,Tian Feng‐Jie¹,Mu Jian‐Bing³,Zheng Jin‐Ping¹⁴^ORCID

Affiliation:

1. Department of Health ToxicologySchool of Public Health, Shanxi Medical University Taiyuan China

2. Department of Biochemistry and Molecular BiologySchool of Preclinical Medicine, Shanxi Medical University Taiyuan China

3. Laboratory of Malaria and Vector ResearchNational Institute of Allergy and Infectious Diseases, National Institute of Health Rockville Maryland USA

4. Department of Public Health and Preventive MedicineChangzhi Medical College Changzhi China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Health, Toxicology and Mutagenesis,Management, Monitoring, Policy and Law,Toxicology,General Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/tox.22932

Reference43 articles.

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4. Transcriptional profiling of the mouse hippocampus supports an NMDAR‐mediated neurotoxic mode of action for benzo[ a ]pyrene

5. Effects of subchronic exposure to benzo[a]pyrene (B[a]P) on learning and memory, and neurotransmitters in male Sprague–Dawley rat

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