Presumptive role of 129 strain-derivedSle16locus in rheumatoid arthritis in a new mouse model with Fcγ receptor type IIb-deficient C57BL/6 genetic background-Reference-Cited by-同舟云学术

Presumptive role of 129 strain-derivedSle16locus in rheumatoid arthritis in a new mouse model with Fcγ receptor type IIb-deficient C57BL/6 genetic background

Published:2011-09-27 Issue:10 Volume:63 Page:2930-2938
ISSN:0004-3591
Container-title:Arthritis & Rheumatism
language:en
Short-container-title:Arthritis & Rheumatism

Author:

Sato-Hayashizaki Aya,Ohtsuji Mareki,Lin Qingshun,Hou Rong,Ohtsuji Naomi,Nishikawa Keiko,Tsurui Hiromichi,Sudo Katsuko,Ono Masao,Izui Shozo,Shirai Toshikazu,Takai Toshiyuki,Nishimura Hiroyuki,Hirose Sachiko

Funder

Grant-in-aid for the Foundation of Strategic Research Projects in Private Universities from the Ministry of Education, Science, Technology, Sports, and Culture of Japan

Research on Intractable Diseases from the Ministry of Health, Labor, and Welfare of Japan

Swiss National Foundation for Scientific Research

Publisher

Wiley

Subject

Pharmacology (medical),Immunology,Rheumatology,Immunology and Allergy

Reference36 articles.

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5. Genetic control of collagen-induced arthritis in a cross with NOD and C57BL/10 mice is dependent on gene regions encoding complement factor 5 and FcγRIIb and is not associated with loci controlling diabetes;Johansson;Eur J Immunol,2001

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