A TGF-β-induced gene, βig-h3, is crucial for the apoptotic disappearance of the medial edge epithelium in palate fusion
Author:
Publisher
Wiley
Subject
Cell Biology,Molecular Biology,Biochemistry
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1002/jcb.22180/fullpdf
Reference31 articles.
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2. The T-box transcription factor gene TBX22 is mutated in X-linked cleft palate and ankyloglossia;Braybrook;Nat Genet,2001
3. Inhibition of TGF-beta 3 (but not TGF-beta 1 or TGF-beta 2) activity prevents normal mouse embryonic palate fusion;Brunet;Int J Dev Biol,1995
4. RGD peptides induce apoptosis by direct caspase-3 activation;Buckley;Nature,1999
5. Overexpression of Smad2 in Tgf-beta3-null mutant mice rescues cleft palate;Cui;Dev Biol,2005
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1. Functions of Matricellular Proteins in Dental Tissues and Their Emerging Roles in Orofacial Tissue Development, Maintenance, and Disease;International Journal of Molecular Sciences;2021-06-21
2. The cleft palate candidate gene BAG6 supports FoxO1 acetylation to promote FasL-mediated apoptosis during palate fusion;Experimental Cell Research;2020-11
3. IRF6 and TAK1 coordinately promote the activation of HIPK2 to stimulate apoptosis during palate fusion;Science Signaling;2019-08-06
4. Activation of Notch1 inhibits medial edge epithelium apoptosis in all-trans retinoic acid-induced cleft palate in mice;Biochemical and Biophysical Research Communications;2016-08
5. IRF6 is the mediator of TGFβ3 during regulation of the epithelial mesenchymal transition and palatal fusion;Scientific Reports;2015-08-04
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