Increased GABAergic function in mouse models of Huntington's disease: Reversal by BDNF-Reference-Cited by-同舟云学术

Increased GABAergic function in mouse models of Huntington's disease: Reversal by BDNF

Published:2004-10-25 Issue:6 Volume:78 Page:855-867
ISSN:0360-4012
Container-title:Journal of Neuroscience Research
language:en
Short-container-title:J. Neurosci. Res.

Author:

Cepeda Carlos,Starling Amaal J.,Wu Nanping,Nguyen Oanh K.,Uzgil Besim,Soda Takahiro,André Veronique M.,Ariano Marjorie A.,Levine Michael S.

Funder

Hereditary Disease Foundation

The Cure HD Initiative

HiQ Foundation

USPHS

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience

Reference77 articles.

1. Neuroprotection by neurotrophins and GDNF family members in the excitotoxic model of Huntington's disease;Alberch;Brain Res Bull,2002

2. Postnatal development of a persistent Na+ current in pyramidal neurons from rat sensorimotor cortex;Alzheimer;J Neurophysiol,1993

3. Striatal neurochemical changes in transgenic models of Huntington's disease;Ariano;J Neurosci Res,2002

4. Amelioration of behavioral deficits in a rat model of Huntington's disease by an excitotoxic lesion to the globus pallidus;Ayalon;Exp Neurol 2004,2004

5. Heterosynaptic dopamine neurotransmission selects sets of corticostriatal terminals;Bamford;Neuron,2004

Cited by 114 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Reduced inhibitory synaptic transmission onto striatopallidal neurons may underlie aging-related motor skill deficits;Neurobiology of Disease;2024-09

2. 14-3-3 proteins—a moonlight protein complex with therapeutic potential in neurological disorder: in-depth review with Alzheimer’s disease;Frontiers in Molecular Biosciences;2024-02-05

3. Thalamic Foxp2 regulates output connectivity and sensory-motor impairments in a model of Huntington’s Disease;Cellular and Molecular Life Sciences;2023-11-21

4. Evidence for glutamate excitotoxicity that occurs before the onset of striatal cell loss and motor symptoms in an ovine Huntington’s Disease model;2023-06-24

5. Striatal Chloride Dysregulation and Impaired GABAergic Signaling Due to Cation-Chloride Cotransporter Dysfunction in Huntington’s Disease;Frontiers in Cellular Neuroscience;2022-01-14