Evidence that Fgf10 contributes to the skeletal and visceral defects of an apert syndrome mouse model

Author:

Hajihosseini Mohammad K.,Duarte Raquel,Pegrum Jean,Donjacour Anne,Lana-Elola Eva,Rice David P.,Sharpe James,Dickson Clive

Publisher

Wiley

Subject

Developmental Biology

Reference51 articles.

1. Apert syndrome mutations in fibroblast growth factor receptor 2 exhibit increased affinity for FGF ligand;Anderson;Hum Mol Genet,1998

2. Fibroblast growth factor 10 (FGF10) and branching morphogenesis in the embryonic mouse lung;Bellusci;Development,1997

3. Rare Mutations of FGFR2 causing Apert syndrome: identification of the first partial gene deletion, and an Alu element insertion from a new family;Bochukova;Human Mutation,2008

4. Crouzon-like craniofacial dysmorphology in the mouse is caused by an insertional mutation at the Fgf3/Fgf4 locus;Carlton;Dev Dyn,1998

5. An updated pediatric perspective on the Apert syndrome;Cohen;Am J Dis Child,1993a

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